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Video Endoscopic Sequence 1 of 2.
Mallory Weiss Tear
This 58 year-old female, 3 days previously had hematemesis.
For more endoscopic details, download the video clip by clicking on the endoscopic image. Wait to be downloaded complete then Press Alt and Enter for full screen. All endoscopic images shown in this Atlas contain video clips. We recommend seeing the video clips in full screen mode.
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Video Endoscopic Sequence 2 of 2.
A Hiatal Hernia is seen with longitudinal tear.
Hiatal hernia has been found in 40 to 100 percent of patients with Mallory-Weiss tears and has been considered by some to be a necessary predisposing factor. It has been proposed that, in hiatus hernia, a higher pressure gradient develops in the hernia compared with that in the rest of the stomach during retching, thereby increasing the potential for mucosal laceration. Gastroesophageal tears may also be more likely to occur when the upper esophageal sphincter does not relax during vomiting.
Mallory-Weiss syndrome is characterized by longitudinal mucosal lacerations (intramural dissections) in the distal esophagus and proximal stomach, which are usually associated with forceful retching. The lacerations often lead to bleeding from submucosal arteries. Since the initial description in 1929 by Mallory and Weiss in 15 alcoholic subjects.
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Video Endoscopic Sequence 1 of 3.
Mallory Weiss Syndrome
This 42 year-old male, two days previously has been drinking alcoholic beverages, started vomiting, immediately patient initiates with hematemesis follow with melena.
The image displays a blood clot that covers the mucosal tear.
The classic presentation consists of an episode of hematemesis following a bout of retching or vomiting, although this presentation may be less common than previously thought.
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Video Endoscopic Sequence 2 of 3.
Retroflexed image
Pathophysiology
A Mallory-Weiss tear (MWT) likely occurs as a result of a large, rapidly occurring, and transient transmural pressure gradient across the region of the gastroesophageal junction. Acute distension of the nondistensible lower esophagus can also produce a linear tear in this region.
With a rapid rise in intragastric pressure due to precipitating factors, such as retching or vomiting, the transmural pressure gradient increases dramatically across the hiatal hernia, which abuts a low intrathoracic pressure zone. If the shearing forces are high enough, a longitudinal laceration eventually occurs. Within the hernia, the tear is more likely to involve the lesser curvature of the gastric cardia, which is relatively immobile compared to the remainder of the stomach.
Another potential mechanism for MWTs is the violent prolapse or intussusception of the upper stomach into the esophagus, as can be witnessed during forceful retching at endoscopy.
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Video Endoscopic Sequence 3 of 3.
The Hiatus Hernia displays the blood clot
- Bleeding from MWTs stops spontaneously in 80-90% of patients. With conservative therapy, most tears heal uneventfully within 48 hours. Thus, a MWT can easily be missed if endoscopy is delayed.
- The degree of blood loss varies. Earlier studies reported that the proportion of patients requiring blood transfusions was 40-70%. These figures do not seem to be the trend today and are probably significantly lower.
- Hemodynamic instability and shock may occur in up to 10% of patients. In one series, mortality as high as 8.6% was attributed to MWTs. Current clinical experience suggests a significantly lower mortality rate from MWTs.
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Video Endoscopic Sequence 1 of 3.
A 68 year-old female, one week previously presented a history of severe vomiting, which later had melena. The endoscopy displayed here is performed one week after the inicial symtoms. A Mallory Weiss tear was showed, presented as a linear ulcer. The Mallory Weiss tear is localized to the gastric side side of the squamocolumnar junction or extends across the z line into the esophagus.
The original description by Mallory and Weiss in 1929 involved patients with persistent retching and vomiting following an alcoholic binge. However, Mallory-Weiss syndrome may occur after any event that provokes a sudden rise in intragastric pressure or gastric prolapse into the esophagus. Pathophysiology: A Mallory-Weiss tear (MWT) likely occurs as a result of a large, rapidly occurring, and transient transmural pressure gradient across the region of the gastroesophageal junction. Acute distension of the nondistensible lower esophagus can also produce a linear tear in this region. With a rapid rise in intragastric pressure due to precipitating factors such as retching or vomiting, the transmural pressure gradient increases dramatically across the hiatal hernia, which abuts a low intrathoracic pressure zone. If the shearing forces are high enough, a longitudinal laceration eventually occurs. Within the hernia, the tear is more likely to involve the lesser curvature of the gastric cardia, which is relatively immobile compared to the remainder of the stomach. Another potential mechanism for MWTs is the violent prolapse or intussusception of the upper stomach into the esophagus, as can be witnessed during forceful retching at endoscopy. Medline.
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Video Endoscopic Sequence 2 of 3.
Same case as above, the retroflexed maneuver displays a linear ulcer at the gastroesophageal junction.
The Mallory Weiss tear is an acute lesion that, when viewed soon after the tear occurs, has the appearance of an edematous and irregular split in the mucosa. Bleeding is usually multifocal, but can arise from an exposed intramural artery branching off the left gastric artery.
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Video Endoscopic Sequence 3 of 3.
The classic presentation for the Mallory Weiss tear is a sequence of events beginning with nausea and vomiting followed soon by hematemesis.
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A 43 year-old man who had been drinking alcoholic beverages, after that, he undergone vomiting and bleeding. The video clip displays a hiatal hernia, reflux esophagitis and a blood clot that covers the mucosal tear.
A history of heavy alcohol use leading to vomiting has been noted in 40 to 80 percent of patients with Mallory-Weiss syndrome in most series. The bleeding is usually more severe when Mallory-Weiss tears are associated with portal hypertension and esophageal varices. Occasionally, patients give a history of ingestion of aspirin or nonsteroidal antiinflammatory drug.
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Mallory Weiss Syndrome.
34 year-old male physician who had been drinking alcoholic beverages and started vomiting, immediately, after that, the patient had an upper gastrointestinal bleeding.
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Video Endoscopic Sequence 1 of 3.
54 year-old female, who had induced vomiting a day before because of a feeling of malaise. Melena was observed. Her hemoglobin was 8.1 mg/dl. She underwent upper endoscopy, and a gastroesophageal tear was found at the gastroesophagic junction.
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Video Endoscopic Sequence 2 of 3.
The image and the video display a ulcer with a blood clot the endoscope is retroflexed.
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Video Endoscopic Sequence 3 of 3.
The blood clot is observed in retroflexed maneuver.
How frequently a Mallory-Weiss tear occurs without bleeding cannot be determined with any certainty. It is highly likely that the condition occurs in a less severe form more frequently than is recognized.
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Mallory-Weiss tears occurring during endoscopy
Mallory-Weiss tears occurring during the course of upper gastrointestinal endoscopy are apparently rare, Iatrogenic Mallory-Weiss tears are rare and generally have a benign course. They tend to occur mostly in patients who have experienced excessive retching or struggling during endoscopy. Mallory-Weiss tears complicating endoscopy occur especially in elderly, female patients with hiatal hernias. The importance of admitting patients with this complication to hospital for overnight observation is recommended in view of the possible development of haemorrhage or perforation.
Pubmed Pubmed
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Video Endoscopic Sequence 1 of 9.
Mallory-Weiss Syndrome
This 54 year-old female, had some episodes of vomiting presented with hematemesis, she was diagnostic as having Mallory Weiss tear, after that the patient presented intermittent melena during a one week, She was referred to our endoscopic unit for evaluation.
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Video Endoscopic Sequence 2 of 9.
An adhered blood clot is observed of where it is the exact site of bleeding.
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Video Endoscopic Sequence 3 of 9.
At the gastric fundus, there are rest of dark blood.
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Video Endoscopic Sequence 4 of 9.
There is a slight bleeding around of the blood clot
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Video Endoscopic Sequence 5 of 9.
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Video Endoscopic Sequence 6 of 9.
Immediately above of the blood clot there is an erosion in phase of healing.
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Video Endoscopic Sequence 7 of 9.
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Video Endoscopic Sequence 8 of 9.
The hemostatic therapy with argon plasma has been initiated
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Video Endoscopic Sequence 9 of 9.
The hemostatic treatment was carried out ambulatorily, the patient was handled with proton pump inhibitors evolving without newness.
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